Texas TPWD CWD positive Panhandle Roadkill Whitetail

January 14, 2018

Texas TPWD CWD positive Panhandle Roadkill Whitetail

http://chronic-wasting-disease.blogspot.com/2018/01/texas-tpwd-cwd-positive-panhandle.html


kind regards, terry

Old news. But, the sky is definitely falling. Thanks for the heads up. I’m gonna go invest in Reynolds wrap.

Yep, all the deer in Texas will soon be gone. Maybe they can figure out a way to get rid of anthrax which is the real threat in certain areas of west Texas.

Terry Singletary — A retired machinist and high school dropout, Terry Singletary suffered the tragic loss of his mother to “sporadic” Creutzfeldt-Jakob disease (CJD) in 1997. Desperate to find an explanation for his mother’s death, he has devoted himself to the sad and fruitless task of connecting her death to her diet. Various reports confirm that Mrs. Singletary’s life was claimed by the most common sub-type of CJD (one that accounts for 70 percent of “sporadic” cases). Sporadic CJD, unlike its newer “variant,” is not linked to meat.

As the self-appointed international coordinator of CJD Watch, an organization he co-founded with social worker Deborah Oney, Singletary is cited in media reports as an apparent expert on tracking mad cow disease. This despite his lack of formal education and the absence for support from any credible academic, medical or scientific authority. His sensationalist allegations about the safety of U.S. beef have found their way into hundreds of newspapers and broadcasts. Singletary moderates a mad-cow discussion forum run by a vegetarian activist group; his contributions account for more than half the traffic on the “BSE-L” mailing list, which is generally read by real scientists. Animal rights activists and other food-scare artists frequently refer to him as “Dr. Terry Singletary,” apparently an honorary degree as he has yet to finish high school.

Like many activists, Singletary ignores overwhelming epidemiological and laboratory evidence that rules out a connection between sporadic CJD and beef. Relying entirely on shallow circumstantial evidence and frequent repetition of claims which have been publicly refuted as false, he also blindly insists upon a mad-cow with Alzheimer’s, Parkinson’s, and Lou Gehrig’s disease. His specific allegations have been clearly refuted by Centers for Disease Countrol and Prevention scientists in the journal Neurology.

I heard that deep frying backstrap prohibits the spread of cwd to humans, but to make sure if you cover with your grandma's gravy there would be no way to contract!

Never let facts or the life's work of hundreds of actual scientists and medical research professionals get in the way of a good story.

LMAO! getting desperate now are ya


i just love it when folks are grasping for straws and fake news trying to cover up the truth. this old article has been proven so terribly wrong, and they just don't get it, still. every thing i said back then has come true...sadly.

SO, just who are The Center for Consumer Freedom ;

http://www.consumerfreedom.com/index.cfm


let's take a closer look shall we ;

The Center for Consumer Freedom (CCF) (formerly called the "Guest Choice Network (GCN)") is a front group for the restaurant, alcohol and tobacco industries. It runs media campaigns which oppose the efforts of scientists, doctors, health advocates, environmentalists and groups like Mothers Against Drunk Driving, calling them "the Nanny Culture -- the growing fraternity of food cops, health care enforcers, anti-meat activists, and meddling bureaucrats who 'know what's best for you.'"

CCF is registered as a tax-exempt, non-profit organization under the IRS code 501(c)(3). Its advisory board is comprised mainly of representatives from the restaurant, meat and alcoholic beverage industries.

http://www.sourcewatch.org/index.php?title=Center_for_Consumer_Freedom

http://en.wikipedia.org/wiki/Center_for_Consumer_Freedom

snip...SEE FULL TEXT HERE ;

http://betaamyloidcjd.blogspot.com/2010/12/alimentary-prion-infections-touch-down.html

Mad Cow Scaremongers by Terry S. Singeltary Sr. a review of the TSE prion agent 2003-2011

http://transmissiblespongiformencephalopathy.blogspot.com/2011/09/mad-cow-scaremongers.html

TUESDAY, DECEMBER 12, 2017

*** Chronic Wasting Disease CWD TSE Prion (aka mad deer disease) Update USA December 14, 2017 ***

(zoonosis and environmental risk factors towards the bottom, after state by state reports)

http://chronic-wasting-disease.blogspot.com/2017/12/chronic-wasting-disease-cwd-tse-prion.html

FRIDAY, DECEMBER 15, 2017

Canada CFIA updating its national CWD TSE PRION efforts to eradicate disease farmed cervid NOT successful December 14, 2017

http://chronic-wasting-disease.blogspot.com/2017/12/canada-cfia-updating-its-national-cwd.html

TUESDAY, DECEMBER 12, 2017

Bovine Spongiform Encephalopathy BSE TSE Prion (aka mad cow disease) Report December 14, 2017 2017

http://bovineprp.blogspot.com/2017/12/bovine-spongiform-encephalopathy-bse.html

MONDAY, JANUARY 1, 2018

PRESIDENT TRUMP KILLS PROPOSED RULE THAT WOULD HAVE PROHIBITED THE USE OF COW BYPRODUCTS IN THE MANUFACTURING OF DRUGS WARNING TO ALL COUNTRIES

http://bseusa.blogspot.com/2018/01/president-trump-kills-proposed-rule.html

TUESDAY, DECEMBER 12, 2017

SCRAPIE TSE PRION UPDATE USA DECEMBER 14, 2017

http://scrapie-usa.blogspot.com/2017/12/scrapie-tse-prion-update-usa-december.html

TUESDAY, DECEMBER 12, 2017

Creutzfeldt Jakob Disease CJD National Prion Disease Pathology Surveillance Center Cases Examined to December 14, 2017

http://creutzfeldt-jakob-disease.blogspot.com/2017/12/creutzfeldt-jakob-disease-cjd-national.html

Tuesday, December 12, 2017

Neuropathology of iatrogenic Creutzfeldt–Jakob disease and immunoassay of French cadaver-sourced growth hormone batches suggest possible transmission of tauopathy and long incubation periods for the transmission of Abeta pathology

http://tauopathies.blogspot.com/2017/12/neuropathology-of-iatrogenic.html


kind regards, terry

O.05: Transmission of prions to primates after extended silent incubation periods: Implications for BSE and scrapie risk assessment in human populations

Emmanuel Comoy, Jacqueline Mikol, Valerie Durand, Sophie Luccantoni, Evelyne Correia, Nathalie Lescoutra, Capucine Dehen, and Jean-Philippe Deslys Atomic Energy Commission; Fontenay-aux-Roses, France

Prion diseases (PD) are the unique neurodegenerative proteinopathies reputed to be transmissible under field conditions since decades. The transmission of Bovine Spongiform Encephalopathy (BSE) to humans evidenced that an animal PD might be zoonotic under appropriate conditions. Contrarily, in the absence of obvious (epidemiological or experimental) elements supporting a transmission or genetic predispositions, PD, like the other proteinopathies, are reputed to occur spontaneously (atpical animal prion strains, sporadic CJD summing 80% of human prion cases). Non-human primate models provided the first evidences supporting the transmissibiity of human prion strains and the zoonotic potential of BSE. Among them, cynomolgus macaques brought major information for BSE risk assessment for human health (Chen, 2014), according to their phylogenetic proximity to humans and extended lifetime. We used this model to assess the zoonotic potential of other animal PD from bovine, ovine and cervid origins even after very long silent incubation periods.

*** We recently observed the direct transmission of a natural classical scrapie isolate to macaque after a 10-year silent incubation period,

***with features similar to some reported for human cases of sporadic CJD, albeit requiring fourfold long incubation than BSE. Scrapie, as recently evoked in humanized mice (Cassard, 2014),

***is the third potentially zoonotic PD (with BSE and L-type BSE),

***thus questioning the origin of human sporadic cases.

We will present an updated panorama of our different transmission studies and discuss the implications of such extended incubation periods on risk assessment of animal PD for human health.

===============

***thus questioning the origin of human sporadic cases***

===============

***our findings suggest that possible transmission risk of H-type BSE to sheep and human. Bioassay will be required to determine whether the PMCA products are infectious to these animals.

==============

https://prion2015.files.wordpress.com/2015/05/prion2015abstracts.pdf

Transmission data also revealed that several scrapie prions propagate in HuPrP-Tg mice with efficiency comparable to that of cattle BSE. While the efficiency of transmission at primary passage was low, subsequent passages resulted in a highly virulent prion disease in both Met129 and Val129 mice. Transmission of the different scrapie isolates in these mice leads to the emergence of prion strain phenotypes that showed similar characteristics to those displayed by MM1 or VV2 sCJD prion. These results demonstrate that scrapie prions have a zoonotic potential and raise new questions about the possible link between animal and human prions.

http://www.tandfonline.com/doi/abs/10.1080/19336896.2016.1163048?journalCode=kprn20

http://www.tandfonline.com/doi/abs/10.1080/19336896.2016.1163048?journalCode=kprn20

RION 2016 TOKYO

Saturday, April 23, 2016

SCRAPIE WS-01: Prion diseases in animals and zoonotic potential 2016

Prion. 10:S15-S21. 2016 ISSN: 1933-6896 printl 1933-690X online

Taylor & Francis

Prion 2016 Animal Prion Disease Workshop Abstracts

WS-01: Prion diseases in animals and zoonotic potential

Juan Maria Torres a, Olivier Andreoletti b, J uan-Carlos Espinosa a. Vincent Beringue c. Patricia Aguilar a,

Natalia Fernandez-Borges a. and Alba Marin-Moreno a

"Centro de Investigacion en Sanidad Animal ( CISA-INIA ). Valdeolmos, Madrid. Spain; b UMR INRA -ENVT 1225 Interactions Holes Agents Pathogenes. ENVT. Toulouse. France: "UR892. Virologie lmmunologie MolécuIaires, Jouy-en-Josas. France

Dietary exposure to bovine spongiform encephalopathy (BSE) contaminated bovine tissues is considered as the origin of variant Creutzfeldt Jakob (vCJD) disease in human. To date, BSE agent is the only recognized zoonotic prion. Despite the variety of Transmissible Spongiform Encephalopathy (TSE) agents that have been circulating for centuries in farmed ruminants there is no apparent epidemiological link between exposure to ruminant products and the occurrence of other form of TSE in human like sporadic Creutzfeldt Jakob Disease (sCJD). However, the zoonotic potential of the diversity of circulating TSE agents has never been systematically assessed. The major issue in experimental assessment of TSEs zoonotic potential lies in the modeling of the ‘species barrier‘, the biological phenomenon that limits TSE agents’ propagation from a species to another. In the last decade, mice genetically engineered to express normal forms of the human prion protein has proved essential in studying human prions pathogenesis and modeling the capacity of TSEs to cross the human species barrier.

To assess the zoonotic potential of prions circulating in farmed ruminants, we study their transmission ability in transgenic mice expressing human PrPC (HuPrP-Tg). Two lines of mice expressing different forms of the human PrPC (129Met or 129Val) are used to determine the role of the Met129Val dimorphism in susceptibility/resistance to the different agents.

These transmission experiments confirm the ability of BSE prions to propagate in 129M- HuPrP-Tg mice and demonstrate that Met129 homozygotes may be susceptible to BSE in sheep or goat to a greater degree than the BSE agent in cattle and that these agents can convey molecular properties and neuropathological indistinguishable from vCJD. However homozygous 129V mice are resistant to all tested BSE derived prions independently of the originating species suggesting a higher transmission barrier for 129V-PrP variant.

Transmission data also revealed that several scrapie prions propagate in HuPrP-Tg mice with efficiency comparable to that of cattle BSE. While the efficiency of transmission at primary passage was low, subsequent passages resulted in a highly virulent prion disease in both Met129 and Val129 mice. Transmission of the different scrapie isolates in these mice leads to the emergence of prion strain phenotypes that showed similar characteristics to those displayed by MM1 or VV2 sCJD prion. These results demonstrate that scrapie prions have a zoonotic potential and raise new questions about the possible link between animal and human prions.

http://www.tandfonline.com/doi/abs/10.1080/19336896.2016.1163048?journalCode=kprn20

why do we not want to do TSE transmission studies on chimpanzees $

5. A positive result from a chimpanzee challenged severly would likely create alarm in some circles even if the result could not be interpreted for man. I have a view that all these agents could be transmitted provided a large enough dose by appropriate routes was given and the animals kept long enough. Until the mechanisms of the species barrier are more clearly understood it might be best to retain that hypothesis.

snip...

R. BRADLEY

BSE INQUIRY REPORT


kind regards, terry

I hunt the panhandle. This will not change anything for me.

If I'm gonna worry about something killing me, that would be cancer, heart disease etc.... Correct me if I'm wrong Terry, but to this date the data shows I have a much greater chance of dying in a commercial plane crash than I do of CWD. I still use commercial flights anyway.....so I'm certainly not gonna stop eating the venison I kill or pay to have it tested before I do.

Going by your logic of a scorched earth solution for CWD, we should ground all commercial airliners from this day forward.

Man, I really enjoyed the break we have had from these idiotic posts. Guess Mr Singletary was on vacation (or in jail) or something, whatever it was is now over, & now it back to [censored].

Haha. You cited Wikipedia as a source. Hilarious

1. I’d rather die almost any other way.
2. The incubation periods in deer are around two years from infection to death, but in the monkey study, they “sacrificed” the monkeys at 4-7yrs from the date of infection. CWD has been detected in TX for only six years, and until recently it was only a handful of cases. There could be a substantial number people infected with CWD before we ever know it. I’m not saying they actually are, but how would we know? Have you been tested?
3. If you read the scrapie studies, and that’s almost certainly where it started, you’ll find that scrapie is contagious to sheep even through skin contact with a cleaned metal post that had been in contact with diseased sheep MONTHS earlier. Other documentation suggests scrapie can survive for up to 16yrs in the environment.

This is not the deer flu where we might suffer some economic loss if X percent of our heard dies. This is a very very scary disease with very high transmission rates, and 100% mortality rates once infected. On top of that, the impact is very long term. If it ever makes the jump to humans, it may be turn out to be much more transmissible than BSE. This is not just a “don’t eat raw brains” situation.

The sky may not be falling, but the people saying this has been around forever and it won’t infect a human are taking a very irresponsible stance with absolutely no science backing it up. It hasn’t been in Texas long enough for us to know if humans can get it, and I’m not aware of humans being tested for it. The best thing about it is that it has been in CO for quite a few years and currently we don’t have a lot of evidence that it has jumped in CO, even if it can’t be ruled out. I’m for maintaining the mandatory checks inside the surveillance zones, testing any roadkill found, testing any deer volunteered to be tested even outside the zones, and continuing to look for a way to stop its spread.

A less far fetched scenario would be that it makes the jump to cattle. Given it’s easy transmission amoung deer via saliva, fecal matter, food sources etc. If it ever did jump to cattle the problem would be crazy.

You and flounder should meet and have lunch.

Maybe. Here are the questions. With a disease that could have a 10+ year incubation time, isn’t regularly tested for by doctors, and was only detected in Texas in 2012, and that was just one animal, how many years will it be before the guy that ate the first one gets diagnosed? How many people will be infected by the time that first guy gets diagnosed? How far will it have spread by then? Will it be in livestock by then?

I’m not saying the sky is falling. We don’t know if it is or not. But I’m not going to bury my head in the sand and say that until we have proof that it will spread to people we should ignore it. We don’t have proof that it will, but we do have evidence that it can, and we certainly don’t have proof that it can’t. Why not research it before it’s out of control?

At least CO has had it for a while and we still don’t see confirmed human cases.

Yes you did imply the “sky is falling” by saying “this is a very scary disease with very high transmission rates”.

There is absolutely no data supporting the lie that it has a high transmission rate. Its been known to be in existence in Colorado and Wyoming for decades and hasn’t decimated any populations.

You also imply that it’s ONLY been in Texas for a few years when in fact no one knows how many years it’s been in ANY state. We just know when cases were found.

1) A lot of things can make something scary other than their likelihood. There are all kinds of snakes, spiders and diseases in South America that are scary, yet very uncommon.
2) 10%-12% disease prevalence in a wild population is a high transmission rate to me. There are some areas where it is that prevalent.
3) how bout I change my position to say “it probably has not been prevelant in TX too much prior to 2012”? Better? Since it’s been known since 1968, I doubt there were very many deer in TX that had it and that far back without anyone noticing till 2012. Obviously they probably didn’t notice the very first one, or even the very first year.
4) None of this means the sky is falling, but you won’t catch me saying we shouldn’t test any more deer or research the disease any further. I think there should be more check stations for VOLUNTARY checks all over the state so that it’s more convenient. I would like to get a deer tested next time I kill one, but since I’m outside the zone, I won’t be driving two hours to drop off a sample. Did you notice that, I won’t drive two hours to have a deer tested. If I thought the sky was falling I’d probably either make the drive or quit hunting all together.

I know squatt about CWD. What I don't understand is that the WT Deer population of Texas is very large. Yet there have been very few confirmed cases of CWD reported.And other than those reported at Deer breeders the supposedly confirmed cases have been many miles apart. They say that Deer will show signs of having it. I have not heard of anyone saying they have observed these signs in Deer while hunting. I hope this CWD scare is not something like global warming where the dire predictions of how bad it is keeps changing when those predictions don't happen each year.

If you're actually worried about deer health/mortality, work on anthrax, internal parasites, hemorrhagic disease, blue tongue, bovine tuberculosis & tick born disease's.
You scientific types know, the things that are easily 'picked up' & actually do kill deer in numbers.

Brain abscesses caused by pedicule injury or by problems associated with abnormal antler shedding probably kill more bucks every year in a handful of Tx counties than CWD does nationwide.
How many have seen a buck or any deer with brain abscess, overload of internal parasites, anthrax or any other fatal ailment.

Do we want to turn a blind eye to CWD, no not at all, but there is no need or advantage to using unsubstantiated overly hyped scare tactics and humongous leaps in relevance wrapped up in a sensationalist 'fake' news/media format most of which is a manipulated BS tidbits version of sound reasonable scientific method.

It certainly could become prevalent in the future, with a little help from its friends.
And if you don't think that happens frequently by those whose funding & entire life's work depends on it, I've got some ocean front property in Kansas to sell.

Dang there are some world class googlers on this thread!

It takes years to incubate, and deer don’t show symptoms till the end of its course. They can transmit it before that. Because it kills slowly, a lot of deer that get it will die of something else(hunting) before they show symptoms, but not before they transmit it to other deer. The cases that pop up many many miles from any other cases problem come from killing a deer with CWD(that you didn’t realize had it) in or near a known CWD area, and transporting that carcass home to a CWD free area. Because the infectious agent can live in the soil for many years, it is almost inevitable that a deer will eventually come into contact with soil where that carcass was dumped if it was dumped out in the open somewhere that deer actually exist.

I hope all it is, is a global warming type scare that never amounts to anything.

So that's where you draw the line on non sense theories? Lose your mind over CWD, but global warming is just crazy. Gotcha.

The globe hadn’t warmed since 1998 until they started revising data and using temps in the wakes of ships which ALWAYS register higher than temps from bouyes. Global warming is a political tool with scientists on both sides of the issue, but politicians on only one side.

I haven’t lost my mind on CWD, I’m just saying we should keep an eye on it. There are a few things about the disease that make it different from others. 100% mortality, long incubation times, persistence in the environment, and a particularly slow uncomfortable death. The long incubation time makes it hard to track, the persistence in the environment make it hard to stop, and the other factors make it bad to get. It’s the perfect hoax disease. I hope it’s a hoax. If it’s not a hoax, what’s wrong with gaining some knowledge about now, instead of when it’s all over? If it’s the worst case scenario, it will probably be a 10-30 years before we know it. What’s wrong with doing a little research now instead of waiting? It is real. No one argues that. What’s wrong with doing a little research on it?

You understand that research has been going on don't you? Just because it "showed up" in Texas recently doesn't mean that it's this brand new diseases that we know nothing about. And if you see global warming as a political stunt, how could you possibly not see that's exactly what is going on with CWD?

Yes I know that it is being studied. Some comments in this thread suggest that it shouldn’t be. I think that it should continue to be studied and that, as with any serious disease steps should be taken to contain it until we know that it doesn’t need to be contained. I like that NM will not allow you to leave a CWD zone with anything but meat, hide and a cleaned and bleached skull plate. I like that Texas requires testing inside of CWD monitoring zones and tests roadkill outside the zones. I wish there were more voluntary testing stations, and those will probably come on their own in time.

I never said that it definitely wasn’t a political stunt. I hope that it is just a political stunt for funding, but I don’t think we really know the answer to that yet. There are two big factions in this debate. The people that think it’s the near inescapable zombie apocalypse, and the people that think it’s all made up crap. A few features of the disease allow it to fit nicely in either category. The long incubation time means that it does not rapidly decimate populations. Many infected deer die of something else before we know they were infected, and because they would have died anyway, their death removed the disease’s impact on the population(compensatory mortality). It also results in a spreading rate that is not only low, but seems even lower than it is. These features make the unbelieving side assume it’s not spreading and isn’t serious. They also make the apocalypse side freak out about how it’s already all over and everything has it. The next feature is the mode of transmission. The disease persists in the environment and seems highly contagious. BSE pretty much required someone/something to eat infected tissue. We stopped feeding cows the leftovers of processed cows and BSE pretty much went away. This disease seems more like scrapie. It lasts a long time in the environment, isn’t destroyed by rendering, and is taken up by plants, so the deer doesn’t have to eat an infected deer. It may have to near an infected deer’s waste, or it may simply have to eat grass where an infected deer’s carcass was disposed of two years ago. So the nay sayers say that deer don’t eat deer, and the fact that it pops up in places isolated from other known areas is proof that it’s been around forever and we’re just now finding it, while the zombie freak outs say that it’s inescapable and going to slowly sweep across the world. I personally think that most of the spreading probably comes from carcass disposal. You shoot a deer in CO, and dump it Medina and bingo. Some probably comes from migration. Those mountain muleys will cover some serious territory from time to time.

I don’t think I fall in either camp, but am certainly closer to the alarmists than the other side. There is some evidence that it can jump species more easily than BSE, and that unlike BSE, which remains BSE even in other hosts, CWD adapts slightly when they infect a new host species with it. That means that if I do shoot a deer in a CWD zone, I will not be eating it until the test comes back negative. I don’t think it has infected hunters yet or we would see more CJD in CO. It hasn’t really been known to be in TX long enough to know, but I bet plenty of people in CO have eaten deer with CWD. It’s been known in CO for 50yrs, so it’s probably not infecting humans. One reason could well be how deer are processed. Beef animals are sawn in half down the center of the spine getting spinal fluid and tissue right on all your good steaks. Most people don’t split a deer like that. I’m not sure how processors do it. I’ve sawn deer in half down the spine, but I won’t be anymore. I do think that that it is spreading from hunters not being careful. I think that it would be wise for states to eliminate carcass movement from areas known to contain CWD(allow only boned out meat, hides and skull plates). But that’s up to individual states. I’m not for eradicating herds, or stoping hunting until we know more, but I’m not for ignoring it either. I think the studies should focus on whether it can be transmitted to other species from eating meat(the macaque study includes that) and then if it can be transmitted from animal to animal without the consumption of meat.

Did it ever occur to you BillT that this disaease has been in the deer population way before you were born on this earth? If we as humans were hunted what percentage of our population could appear to be healthy but have cancer? We are still here, more of us than there have ever been on this earth. There are more deer now than there were when the pilgrims landed. Should we quarantine all the folks in Texas that are diagnosed with cancer? Stop burying all those that die from it because it could spread into the soil?

Yes it did occur to me, but then I did some reading on the subject. CWD is most likely a mutated form of scrapie that probably came from housing deer in pens that had been used to study scrapie. Unlike cancer, which is not contagious, CWD is transmissible from one deer to another deer. We have a group of people ignoring the disease and a group of people freaking out, and they’re ignoring each other instead of trying to get down to the facts.

It has not been proven cancer is not contagious. There are numerous theories that it could be started by a bacteria or virus that attacks our cells. Most agree that the tumors aren't contagious, but know one knows for sure what starts the cell mutation that turns into tumors that kill or kills other cells.

You are assuming a lot of things to be factual when you post about CWD that are not facts at all, just assumptions. You know what happens when you assume, right?

I’m not aware of it being a cancer virus that causes cancer, so much as it is individual reactions and mutations that result from more common infections. Of course there are lots of types of cancer, some with known causes, some with unknown causes. Benzene has been proven to cause kidney cancer. Not all kidney cancer comes from benzene exposure. Certain forms of papilloma virus do result in cancer at fairly high rates, and vaccines have been developed to prevent infection from those viruses. None the less, I’m not aware of any form of cancer which has been shown to directly spread from person to person. There are also various treatments for cancer and very few cancers have 100% mortality rates. So far with CWD we have identified the infectious agent and it can be tested for. CWD has been proven to be directly transmissible from deer to deer, which I’m not aware of in the case of cancer. CWD has been detected at rates of over 10% in some wild populations, but I’m not aware of any cities anywhere where 10% of people currently have cancer. BSE was essentially stopped in its tracks fairly quickly once it was taken seriously. I’m not saying we need to eliminate herds or stop hunting or quarantine states. I’m saying we need to keep studying the disease, take a few sensible precautions(like test your deer, and don’t eat it if it’s positive. CO will issue you a new tag if your deer tests positive, leave your carcass where you shot it if it’s in a CWD containment area) and when we know more about it, then we can change our strategy based on that information. We may find that it’s older than we thought and just won’t mutate and can’t possibly infect anything but cervids and it’s been everywhere for centuries and it just doesn’t matter. Why would you assume that though? I’m not saying the sky is falling. I’m saying that cloud over there could be a storm cloud. You can keep fishing and check that cloud every hour or two, and if it gets bigger and closer you might want to fish within an hour of shore, or you can ignore the cloud and find yourself two hours off the coast when the weather gets rough. There’s a cloud out there. Don’t pack it in and find a new career, but don’t go below deck and take a nap either. Keep your eyes on it and act accordingly. There will always be clouds, and that’s not a reason to panic, but it doesn’t mean a storm will never come either

. I have some swollen lymph nodes that have been swollen for years. I also handle chemicals. I had a doctor take a look and he said there is definitely something there, but I don’t have any other signs that I should be concerned about lymphoma. The recommendation was to have a blood panel and physical exam once a year and keep an eye on it. It will most likely never amount to anything, but it could, and it would be better to know early. I could skip the exam and blood panels and wait until I had obvious symptoms, but by then the it would be a bigger deal than if it was caught at an earlier stage. We already have the sign that something is there. It’s probably not time to treat for lymphoma, but it would be stupid not to get the annual exam and blood work.

Why are you so sure that it’s been around forever and never mattered and therefore never will matter? Viruses mutate, bacteria mutate, crayfish mutate. Why can’t a scrapie prion mutate?

I'm not sure it's been around forever. My point I'm trying to make is we have bigger fish to fry. There are known diseases that kill off entire deer populations already mentioned in these threads. Nothing has been done to contain these higher risks to deer populations. (Blue Tongue, Anthrax) So ask yourself why not? Remember that people like flounder and his supporters want funding for their research. There are other forces at work that have used this disease for political purposes like shutting down deer breeders and high fence hunting operations without any proven facts to support the actions they have taken killing thousands of deer without any positive result to show for it. If flounder was able to do what he wants, he would kill every deer in all the areas CWD has been found. Also remove all the soil, farm and ranch equipment. Even the furniture.

If you don't believe me, ask him your self.

As for me assuming, I don't assume anything about the disease. I just use logic based on the facts I know to be true.

Deer have been found dead of natural causes for as long as they have been in existence. Logic tells me someone hunting them has eaten deer that were carrying a disease that eventually would be fatal to them but outward they appear quite normal. Logic tells me for this reason humans have very likely eaten deer infected with CWD. I could of been one of them. That's why I'm not concerned about contracting CWD from deer I eat. I will continue to use common sense, being that if I kill a deer that doesn't appear to be healthy I won't eat it. I've always managed wild game this way. Years before we were talking about CWD.

Look at the incredible amount of $ and resources being spent on CWD. Compare it to the amount being spent on anthrax (a disease KNOWN to kill people and that can and does kill up to 90% of deer in a single outbreak).

The disparity in THAT number is all I need to know about the political feather lining sky is falling bullch#@ message of people like Flounder and billit.

I say we start human testing for this....lets start with Bill and Terry, with the old method.

TUESDAY, FEBRUARY 13, 2018

MISSISSIPPI STATE DEPARTMENT OF HEALTH Chronic Wasting Disease: Public Health Recommendations

http://chronic-wasting-disease.blogspot.com/2018/02/mississippi-state-department-of-health.html


kind regards, terry

This has to be one of the most stupid recommendations I've ever seen! Logic has left the state of Mississippi.....or maybe it was never there to start with.

As I said on the Wisconsin thread. The "how ignorant can I be" sweepstakes are just getting started. We will have soooo many entertainment ops in the future. I'm stocking up on cigars and scotch. He77 with popcorn.

Oh, and if you think tpwd is any smarter or immune to the ignorance.... in my best Ron White voice "You're WROOOOOONG"!!

I’m not as extreme as Flounder on the issue. I have however read a lot of the links he’s posted as well as a lot of the not so technical writings saying not to worry about CWD, and I have formed my opinion based on my findings. Like I said in an earlier post, you don’t have to believe everything Flounder says about the impending zombie apocalypse or else believe that CWD is s meaningless waste of time, but those seem to be the two options most people in these threads give. My opinion, after reading info from both sides, is that you should at least pay attention to it. Personally, I won’t be eating any deer that I kill in or near a CWD zone until I receive a negative test result. The only deer I hunt near any of those areas involve packing the animal out, so I bone them out anyway. Why not pay a few bucks to have it tested for peace of mind?

For purpose of argument, if there were an outbreak of anthrax or blue tongue etc. would any of you be ok with a requirement that carcasses within a containment zone be left within the containment zone? The answer does not have to apply to CWD, just wondering if there is a disease that justifies any form of restrictions.

For purpose of argument, if there were an outbreak of anthrax or blue tongue etc. would any of you be ok with a requirement that carcasses within a containment zone be left within the containment zone? The answer does not have to apply to CWD, just wondering if there is a disease that justifies any form of restrictions.

In neither case is transmission of these diseases facilitated by "moving carcasses". The vector is not the carcass. So why would you restrict something that isn't going to have any effect??

Well, that's an easy question to answer. You do that in order to show your chicken little constituents/colleagues that you are "doing something".

Research for vaccines/treatments for anthrax/ehd would be supported. And should be a much higher priority than research on a disease that has virtually no effect on populations and NO effect on human health.

Think about it, anthrax will kill you in a heartbeat. It kills most mammals that are exposed to it. When an outbreak occurs it can kill almost every deer in the affected area. Although it's not deer season when it typically blooms, pigs and exotics are killed and consumed by the tens of thousand of tons during those outbreaks. And when was the last time you heard of someone dying of anthrax from handling a carcass or consuming the meat????

Anthrax is EASILY transmitted from deer to deer. CWD can only be transmitted to primates by incredibly sophisticated and complex methods. So, why the ridiculously disproportionate concern over CWD?

Well, would you be ok with a restriction that actually would reduce transmission?

The Canadian study on macaques included simply eating infected muscle tissue. That’s not exactly an incredibly sophisticated and complex method. You probably can’t get it from a eating meat from a preclinical infected deer. I don’t know how much was proven on the transmission of BSE before they solved the problem buy just keeping BSE from developing in cattle before we eat them, but I would guess that human transmission was related to the fact that cattle are cut in half and spinal fluid and tissue end up getting on all your steaks. I won’t be cutting any more deer in half. I’ll have my venison steaks boneless from now on. With a disease with a very long incubation time, by the time you find out it has infected a human, it will have infected LOTS of humans. It could be decades before the first infected person finds out. Personally, if I shoot a deer in a known CWD area I’m going to have it tested before I eat it, and won’t eat one that tests positive. One deer that don’t test positive or aren’t tested, I will avoid lymph nodes and won’t split them in half.

(xi.) There was concern about contamination during carcase splitting and, in particular, concern that splitting often caused the spinal cord to be severed which could result in small pieces of cord becoming lodged between vertebrae;[14]

(xii.) There was concern about mechanically recovered meat, particularly that recovered from the spinal column;[15]

(xiii.) There was concern that removal of the specified offals did not fully remove the nervous and lymphatic tissue from the animal leaving some of those tissues in food for human consumption;[16]

BSE Inquiry

DFA 15 Monitoring and Enforcement of the SBO Specified Bovine Offal Regulations

http://bseinquiry.blogspot.com/2017/08/dfa-15-monitoring-and-enforcement-of.html


kind regards, terry

So. You don’t see anything to be concerned with about a “study” that supposedly had monkeys getting infected from eating muscle tissue when we know the prions aren’t located in muscle tissue?

I’m fairly certain the statement you made here is incorrect. My understanding is that while the prions are concentrated in nervous and lymphatic tissues, there are some prions located in all tissues. You also have to realize that all muscles contain nerves, or we couldn’t move. Also, processing can get spinal fluid and tissue on your meat, so eating muscle, which is supposed to have very few prions, could actually expose you to very high numbers of prions. The people who got BSE weren’t eating brains, they were eating muscle that was probably contaminated during processing. Although it seems to be transmissible via certain methods with very few prions, oral transmission, as far as I know, does require more prions. If you/your processor don’t split the deer in half during processing and avoid lymph nodes and such, you are probably reducing risk considerably. I’m also probably going to start getting all the meat off the carcass before I remove the head as well. Because I dress skin and quarter in the field already, and completely debone if I’m packed in, it’s really no extra trouble for me to do it that way. I used to finish caping and get the neck meat back at the house, but I’ll just do that in the field now.

I don’t know how they processed the meat for the study, and what I read on it did not specify the condition of the whitetail whose meat was used. I’m sure a dead or near dead wasting deer is going to have far more prions in its meat than a seemingly healthy deer that’s been infected for only a few months. Whatever the case, the disease is hoping around the country, so I’m just gonna make sure I handle my deer in a way that will reduce any possible exposure.

FRIDAY, FEBRUARY 16, 2018

Texas Deer Breeders Continue fight against the state’s wildlife agency and its regulations trying to contain CWD TSE Prion

http://chronic-wasting-disease.blogspot.com/2018/02/texas-deer-breeders-continue-fight.html


kind regards, terry

wrong again...see;


Prion Infectivity in Fat of Deer with Chronic Wasting Disease▿

Brent Race#, Kimberly Meade-White#, Richard Race and Bruce Chesebro* + Author Affiliations

In mice, prion infectivity was recently detected in fat. Since ruminant fat is consumed by humans and fed to animals, we determined infectivity titers in fat from two CWD-infected deer. Deer fat devoid of muscle contained low levels of CWD infectivity and might be a risk factor for prion infection of other species.

http://jvi.asm.org/content/83/18/9608.full


Prions in Skeletal Muscles of Deer with Chronic Wasting Disease

Here bioassays in transgenic mice expressing cervid prion protein revealed the presence of infectious prions in skeletal muscles of CWD-infected deer, demonstrating that humans consuming or handling meat from CWD-infected deer are at risk to prion exposure.

http://science.sciencemag.org/content/311/5764/1117.long


*** WDA 2016 NEW YORK ***

We found that CWD adapts to a new host more readily than BSE and that human PrP was unexpectedly prone to misfolding by CWD prions. In addition, we investigated the role of specific regions of the bovine, deer and human PrP protein in resistance to conversion by prions from another species. We have concluded that the human protein has a region that confers unusual susceptibility to conversion by CWD prions.

Wildlife Disease Risk Communication Research Contributes to Wildlife Trust Administration Exploring perceptions about chronic wasting disease risks among wildlife and agriculture professionals and stakeholders

http://www.wda2016.org/uploads/5/8/6/1/58613359/wda_2016_conference_proceedings_low_res.pdf



kind regards, terry

FRIDAY, FEBRUARY 16, 2018

Wisconsin Stop private deer industry from trucking CWD across state

https://www.greenbaypressgazette.com/sto...tate/342532002/

http://chronic-wasting-disease.blogspot.com/2018/02/wisconsin-stop-private-deer-industry.html


kind regards, terry

You need your own forum for this BS

Please. The ignore button doesn't cover new threads.

FRIDAY, FEBRUARY 16, 2018

Wisconsin Deer from Now-Quarantined PA Lancaster County Farm Tests Positive for Chronic Wasting Disease CWD TSE Prion

http://chronic-wasting-disease.blogspot.com/2018/02/wisconsin-deer-from-now-quarantined-pa.html


kind regards, terry

That study isn’t peer reviewed or published.

Here:
http://www.myewa.org/blog/fake-chronic-wasting-disease-cwd/

https://pdfs.semanticscholar.org/f6f5/99c913fc382f308b1df383633aca51e52065.pdf


but recent investigations have also demonstrated the presence of PrP d in skeletal muscles of TSE-infected laboratory mice and hamsters, of sheep, deer and humans ( Bosque et al., 2002 ; Bartz et al., 2003 ; Glatzel et al., 2003 ; Thomzig et al., 2003 ; Andréoletti et al., 2004 ; Mulcahy et al., 2004 ; Ashwath et al., 2005 ; Casalone et al., 2005 ; Angers et al., 2006 ; Thomzig et al., 2006 ). The levels of PrP d that accumulated in muscles were lower than those found in CNS or lymphoid tissue; however, in rodents, muscle-derived PrP d was shown to be infectious ( Bosque et al., 2002 ; Angers et al., 2006 ). The detection of PrP d (designated PrP Sc in scrapie) in skeletal muscles of scrapie-infected sheep ( Andréoletti et al., 2004 ) and of infectivity in skeletal muscle of CWD-infected mule deer ( Angers et al., 2006 ) has raised the level of concern on the issue of potential human health risks that might be encountered by consuming prion-containing meat.

http://jgv.microbiologyresearch.org/content/journal/jgv/10.1099/vir.0.81777-0#tab2


Our tissue blot findings in skeletal muscles from CWD-infected
WTD would be consistent with an anterograde spread of
CWD prions via motor nerve fibres to muscle tissue (figure 4A).
Similar neural spreading pathways of muscle infection were
previously found in hamsters orally challenged with scrapie [28]
and suggested by the detection of PrPTSE in muscle fibres and
muscle-associated nerve fascicles of clinically-ill non-human
primates challenged with BSE prions [29]. Whether the absence
of detectable PrPTSE in myofibers observed in our study is a
specific feature of CWD in WTD, or was due to a pre- or
subclinical stage of infection in the examined animals, remains to
be established. In any case, our observations support previous
findings suggesting the precautionary prevention of muscle tissue
from CWD-infected WTD in the human diet, and highlight the
need to comprehensively elucidate of whether CWD may be
transmissible to humans. While the understanding of TSEs in
cervids has made substantial progress during the past few years,
the assessment and management of risks possibly emanating from
prions in skeletal muscles of CWD-infected cervids requires further
research.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3069970/pdf/pone.0018345.pdf


kind regards, terry

Terry,
It would probably serve you well to read the link I posted. According to one of your buds approx 7000-15000 infected deer are consumed by humans each year. IF that is anywhere close to true then we have 50 years of data in the largest sample pool ever shouting that humans simply can’t be infected by eating deer meat.

Funny how we keep getting fired up over information that is posted from blogs, and not from actual scientifically backed cites.

Clayton Wolf? Is that you?

Clayton knows better.

it would serve no one, nothing to read the junk science you shooting pens are putting out when it comes to cwd tse prion therancher.


> However, to date, no CWD infections have been reported in people.

key word here is 'reported'. science has shown that CWD in humans will look like sporadic CJD. SO, how can one assume that CWD has not already transmitted to humans? they can't, and it's as simple as that. from all recorded science to date, CWD has already transmitted to humans, and it's being misdiagnosed as sporadic CJD. ...terry

LOOKING FOR CWD IN HUMANS AS nvCJD or as an ATYPICAL CJD, LOOKING IN ALL THE WRONG PLACES $$$

*** These results would seem to suggest that CWD does indeed have zoonotic potential, at least as judged by the compatibility of CWD prions and their human PrPC target. Furthermore, extrapolation from this simple in vitro assay suggests that if zoonotic CWD occurred, it would most likely effect those of the PRNP codon 129-MM genotype and that the PrPres type would be similar to that found in the most common subtype of sCJD (MM1).***

http://www.tandfonline.com/doi/full/10.4161/pri.28124?src=recsys

http://www.tandfonline.com/doi/pdf/10.4161/pri.28124?needAccess=true

http://wwwnc.cdc.gov/eid/article/20/1/13-0858_article.htm

TUESDAY, SEPTEMBER 12, 2017

CDC Now Recommends Strongly consider having the deer or elk tested for CWD before you eat the meat

http://chronic-wasting-disease.blogspot.com/2017/09/cdc-now-recommends-strongly-consider.html

SATURDAY, JANUARY 27, 2018

CDC CHRONIC WASTING DISEASE CWD TSE PRION UPDATE REPORT USA JANUARY 2018

http://chronic-wasting-disease.blogspot.com/2018/01/cdc-chronic-wasting-disease-cwd-tse.html

Diagnosis and Reporting of Creutzfeldt-Jakob Disease

Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA Diagnosis and Reporting of Creutzfeldt-Jakob Disease

To the Editor:

In their Research Letter, Dr Gibbons and colleagues1 reported that the annual US death rate due to Creutzfeldt-Jakob disease (CJD) has been stable since 1985. These estimates, however, are based only on reported cases, and do not include misdiagnosed or preclinical cases. It seems to me that misdiagnosis alone would drastically change these figures. An unknown number of persons with a diagnosis of Alzheimer disease in fact may have CJD, although only a small number of these patients receive the postmortem examination necessary to make this diagnosis. Furthermore, only a few states have made CJD reportable. Human and animal transmissible spongiform encephalopathies should be reportable nationwide and internationally.

Terry S. Singeltary, Sr Bacliff, Tex

1. Gibbons RV, Holman RC, Belay ED, Schonberger LB. Creutzfeldt-Jakob disease in the United States: 1979-1998. JAMA. 2000;284:2322-2323.

http://jama.jamanetwork.com/article.aspx?articleid=1031186


terry

Terry, I hear that CNN is hiring. Your fake news would fit in perfect there.

Wake me up when a human ACTUALLY CONTRACTS CWD.